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SPECKLEPEDIA

The largest online database on clinical speckle tracking examples
  • About us
  • Clinical examples
    • Normal example
    • Ischemic heart disease
      • Myocardial infarction LAD
      • Myocardial infarction RCx
      • Myocardial infarction RCA
    • Left ventricular hypertrophy
      • Athletes heart
      • Hypertension
      • Aortic valve stenosis
      • Restrictive Cardiomyopathy (Amyloidosis)
      • Hypertrophic cardiomyopathy (HCM)
      • Fabry
    • Cardiomyopathy
      • Dilated cardiomypathy (DCM)
      • Restrictive Cardiomyopathy (Amyloidosis)
      • Hypertrophic cardiomyopathy (HCM)
      • Non compaction cardiomyopathy
      • Arrythmogenic cardiomyopathy
    • Miscellaneous
      • Myocarditis
      • Pericarditis
      • Sarcoidosis
      • Chemotherapy induced cardiomyopathy
      • Post radiation dysfunction
      • Tako Tsubo cardiomyopathy
      • (Rejection) heart transplant
      • LBBB
      • Intramyocardial tumor
  • Technical aspects
  • Relevant literature

Sarcoidosis

Home Clinical examples Miscellaneous Sarcoidosis

5 key points

  • Due to the patchy distribution in the myocardium, regional abnormal strain patterns and values are seen, not related to coronary artery territory and often multiple regions in the LV and/or RV are affected
  • Reduced circumferential and longitudinal strain correspond to deleayed enhancement on CMR, radial strain remains normal.
  • GLS is typically reduced in patients with cardiac involvement, even when LVEF is preserved and no delayed enhancement on CMR is seen
  • A cut off value of minus 17.3% predicts all-cause mortality, heart failure hospitalization, device implantation, new arrhythmias, or future development of CS on advanced cardiac imaging modalities
  • Possibly, reduced strain in segments where no delayed enhancement is seen on CMR could reflect areas with active disease without irreversible fibrosis.

 

Cases:

  • Sarcoidosis case 1
  • Sarcoidosis case 2

 

 

References

Aggeli C, Felekos I, Tousoulis D, Gialafos E, Rapti A, Stefanadis C. Myocardial mechanics for the early detection of cardiac sarcoidosis. Int J Cardiol. 2013 Oct 12;168(5):4820-1.

Kul S, Ozcelik HK, Uyarel H, Karakus G, Guvenc TS, Yalcınsoy M, Asoglu E,Kemik AS, Tasal A, Gungor S, Karaarslan E, Kart L, Goktekin O. Diagnostic value of strain echocardiography, galectin-3, and tenascin-C levels for the identification of patients with pulmonary and cardiac sarcoidosis. Lung. 2014 Aug;192(4):533-42.

Joyce E, Ninaber MK, Katsanos S, Debonnaire P, Kamperidis V, Bax JJ, Taube C, Delgado V, Ajmone Marsan N. Subclinical left ventricular dysfunction by echocardiographic speckle-tracking strain analysis relates to outcome in sarcoidosis. Eur J Heart Fail. 2015 Jan;17(1):51-62.

Orii M, Hirata K, Tanimoto T, Shiono Y, Shimamura K, Yamano T, Ino Y, Yamaguchi T, Kubo T, Tanaka A, Imanishi T, Akasaka T. Myocardial Damage Detected by Two-Dimensional Speckle-Tracking Echocardiography in Patients with Extracardiac Sarcoidosis: Comparison with Magnetic Resonance Imaging. J Am Soc Echocardiogr. 2015 Jun;28(6):683-91.

Tigen K, Sunbul M, Karaahmet T, Tasar O, Dundar C, Yalcinsoy M, Takir M, Akkaya E. Early Detection of Bi-ventricular and Atrial Mechanical Dysfunction Using Two-Dimensional Speckle Tracking Echocardiography in Patients with Sarcoidosis. Lung. 2015 Oct;193(5):669-75. **

Saeed IM, Coggins T, Main ML, Bateman TM. Cardiac sarcoidosis with visually normal wall motion: role of cardiac MRI, FDG PET, and strain echocardiography. Case report. Eur Heart J Cardiovasc Imaging. 2015 Nov;16(11):1275.

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